New study shows heart damage after COVID vaccine shot

Many people may have died because of wide use of COVID mRNA vaccines.

Below are some excerpts from “Myocarditis with ventricular tachycardia following bivalent COVID-19 mRNA vaccination.”

The authors report this man decompensated within a day of his fifth shot and required defibrillation, mechanical ventilation, and full life support measures for myocarditis which precipitated the cardiac arrest, conduction defects, and heart failure. He stayed in the hospital over a month. The patient was an 81-year-old man.

“Two weeks before his fifth COVID-19 vaccination, no worsening of his heart failure was detected at our regular outpatient clinic. However, on the day following bivalent BNT162b2 (wild and BA.4-5) vaccination (Pfizer–BioNTech), he was rushed to our hospital with dyspnea.”

“This report indicates the need to suspect myocarditis based on clinical presentation and the importance of multimodality diagnosis using electrocardiography, echocardiography, laboratory testing, myocardial scintigraphy, and CMR [cardiac magnetic resonance]. In our case, CMR showed LGE in the inferolateral segments of the epicardial to mid layers, which has been reported to be a characteristic finding in patient with mRNA vaccine-associated myocarditis. Endocardial biopsy is the gold standard for detecting myocarditis but is invasive and thought to have less sensitivity in disorders resulting from epicardial and patchy diseases such as myocarditis. On the other hand, CMR is considered to be the cornerstone for diagnosis of vaccine-associated myocarditis due to its high diagnostic performance, with a reported sensitivity of 88% and specificity of 96% in community-acquired myocarditis. The COVID-19 vaccine is thought to cause myocarditis via direct damage by free spike protein and induction of inflammatory cytokines (e.g., IL-1β and IL-6) by the lipid nanoparticles covering the mRNA. Expression of free spike protein may increase after the initial bivalent vaccination because antibodies against the spike protein of the BA.4-5 variant are yet to be generated. In autopsy cases, histology has shown patchy interstitial myocardial T-lymphocytic infiltration (T-cell dominant; CD4>>CD8) associated with damage to myocytes.⁶ Molecular mimicry between myocyte tissue and the SARS-COV2 spike protein may also produce an anti-myocytic immune response.⁶ Therefore, T lymphocyte-mediated cell injury and heart-specific autoimmunity have been suggested as mechanisms of post-vaccine myocarditis.^”

“The COVID-19 vaccine is thought to cause myocarditis via direct damage by free spike protein and induction of inflammatory cytokines (e.g., IL-1β and IL-6) by the lipid nanoparticles covering the mRNA. Expression of free spike protein may increase after the initial bivalent vaccination because antibodies against the spike protein of the BA.4-5 variant are yet to be generated.”

The highly regarded Dr. Peter A. McCullough made this obervation about the new findings:

“I wonder how many elderly patients have died within a few days of the COVID-19 vaccine, unrecognized and not reported by families, doctors, or others. Only all-cause mortality data published in the coming months to years will give us a clue. In the meantime, all seniors should understand that even if prior shots were tolerated, the next one could be fatal.”

And there has been many findings of young people dying from cardiac problems post vaccine shots.